Tooth remineralization timeline: what to actually expect
Remineralization is real but slower than most marketing implies. Here is the honest timeline based on clinical research, what changes by week, and the warning signs that you need professional help instead.
Measurable enamel changes show up in clinical studies after several weeks of daily use. Remineralization does not whiten teeth: it never changes their shade. Deeper or long-standing white spots often change little at all. Cavitated holes and deep erosion cannot be remineralized at home and need professional treatment. Enamel is 97 percent hydroxyapatite, and rebuilding only works while the rod structure is still intact. Use a nano-HAp paste twice daily and chew xylitol gum after meals.
This guide is by Minvelle. For the exact window this article describes we make a remineralizing gum, 5.7 mg nano-hydroxyapatite per piece, one piece a day, dose published.
Clinical studies on hydroxyapatite toothpastes measure early enamel changes after several weeks of daily use; xylitol replaces sugar, it does not deposit mineral. Remineralization does not whiten teeth: it never changes their shade. Deeper or long-standing white spots often change little at all.
Late-stage damage (cavities, deep erosion) requires professional treatment, not remineralization products. Set expectations accordingly.
The timeline runs on what you do between brushings
Your toothpaste covers 4 minutes a day. Minvelle is a nano-hydroxyapatite chewing gum with xylitol and Chios mastic, designed for the time between meals when enamel is most exposed to dietary acids. Austrian brand, full ingredient list and batch certificate published.
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"Remineralization" gets used to mean almost anything in oral care marketing, which is part of why expectations are so often miscalibrated. In clinical dentistry, the word has a narrower meaning: the deposition of calcium and phosphate ions back into the crystalline structure of enamel that has lost minerals to acid attack, but has not yet collapsed into a frank cavity. That last clause matters more than anything else in this article.
If you bought a remineralizing toothpaste, mouthwash, or gum and you are wondering when, exactly, you should start seeing something, the honest answer is: it depends, and most of the popular timelines are wrong in the same direction. They overpromise on speed and underpromise on consistency. The actual clinical literature tells a more useful story, one with a clearer week-by-week shape and a clearer list of red flags that mean you should stop self-treating and call a dentist.
Remineralization of early enamel damage takes weeks to months of daily care, not days. Lab studies show surface changes within weeks, but clinical trials of hydroxyapatite toothpaste run for months of daily use. Early white spots can partly reverse or stop growing; a cavity with a hole never does. Whatever you use, judge it over months, keep brushing twice a day, and let your dentist track any spots.
| What was studied | Time frame | Result |
|---|---|---|
| Enamel samples in the lab | Days to weeks | Surface changes only, not proof for your mouth |
| Early white spots between teeth (nano-hydroxyapatite gel) | 6 months of use, X-rays at 1 and 2 years | About 37% to 69% of lesions remineralized at 1 year, by group |
| Caries-active orthodontic patients (hydroxyapatite paste) | 6 months, twice daily | No worse than fluoride paste |
| Young children (hydroxyapatite paste) | 1 year, daily | No worse than fluoride paste |
| A cavity with a hole | Never reverses | Needs a dentist |
How long until I see results from remineralizing gum?
For the mechanism behind these timelines, see how enamel remineralization actually works.
Expect weeks to months, not days. Surface enamel hardening in laboratory remineralization studies is measured over a few weeks, depending on the model.
How long does hydroxyapatite take to work?
How long does hydroxyapatite take to work? In the clinical trials, the measured results came after months of daily use, not days: six months, one year and longer. Short lab studies show surface changes within weeks, but that is enamel in a dish, not your mouth.
Here is what the human trials actually measured, and over what time. In highly caries-prone orthodontic patients, a fluoride-free hydroxyapatite toothpaste used twice daily was checked against a fluoride paste over 6 months, with enamel caries progression as the outcome; it performed no worse (Schlagenhauf et al., 2019). In young children, the same kind of comparison ran for a full year of daily brushing (Paszynska et al., 2021). For early white-spot lesions between teeth, adults used a nano-hydroxyapatite gel at home for 6 months, and the lesions were read on X-rays at 1 and 2 years; at the one-year check, between about 37% and 69% of lesions showed remineralization, depending on the treatment group (Grocholewicz et al., 2020).
What about white spots you can see? Early white-spot lesions are the most reversible stage of enamel damage, but visible change is slow. Trials that track them use months as the unit, and even then not every spot improves. Some fade, some stay stable, and stable is already a good result, because it means the lesion stopped growing. Deep or long-standing white spots often change little at all.
The plain takeaway: every study that showed a benefit used hydroxyapatite daily over weeks and months, usually twice a day in a toothpaste. None of these trials tested a gum, and none tested Minvelle. So we make no timeline promise for our gum. If you use one, think of it as a daily habit, chewed for at least 20 minutes after meals, next to your normal brushing, and judge it over months, not the first week. A dentist can tell you whether a spot is getting smaller, bigger or staying put.
What remineralization can and cannot fix
Enamel is roughly 97% hydroxyapatite by weight, arranged in tightly packed prisms. When the local pH in the mouth drops below 5.5 (the critical demineralization threshold widely cited in caries research), calcium and phosphate begin dissolving out of the surface and subsurface enamel. If saliva, fluoride, hydroxyapatite, or other ion-donating sources are present, the process can run in reverse, and crystals can rebuild from solution. This back-and-forth happens every day in everyone's mouth. A "carious lesion" only develops when demineralization outpaces remineralization for long enough.
So what can remineralization actually fix? In short, anything where the enamel rod structure is still intact but partially demineralized. Clinically this is called an "incipient lesion" or a "white spot lesion": the chalky, dull-white patches you sometimes see on a front tooth after braces come off, or along the gumline after a few weeks of inconsistent plaque control. Studies in dental research journals have reported that early lesions can rehard and regain mineral content under remineralizing protocols in lab settings.
What it cannot fix is a cavitated lesion: a tooth that has lost enamel volume, where the surface has caved inward and bacteria are now occupying the cavity itself. The protein scaffold that originally guided enamel formation (the ameloblast cell layer) is gone by the time teeth erupt. The body cannot rebuild missing enamel volume the way it rebuilds bone, because adult enamel is not a living tissue. Once you have a hole, you need a filling.
Reversible vs not. If the surface still feels smooth to a dental explorer and looks intact (even if discolored or chalky), it is likely remineralizable. If the explorer catches in a soft pit, or you can see a defined dark hole, that is cavitated. No amount of toothpaste, gum, or rinse will refill it.
There is also a borderline zone (sometimes called "microcavitation") where the surface has begun to break down at a microscopic scale but has not yet formed a clinically visible cavity. Some of these lesions can still arrest under aggressive remineralization with frequent fluoride or hydroxyapatite exposure. Others progress. This is the zone where a dentist's judgment, not a consumer product, makes the call.
Week-by-week timeline: what to expect
The honest timeline is built on two facts. First, the mechanisms of remineralization operate on different layers of the tooth (cementum, dentin, enamel surface, enamel subsurface) at different rates. Second, what you can perceive (look, feel) lags the underlying mineral changes by weeks. The grid below summarizes what most clinical studies on hydroxyapatite, xylitol, and fluoride agents have found about the rough sequence of changes.
A few things stand out from this sequence. Visible changes in early white spots lag because they require actual crystal reorganization, which is slow. And the gap between "measurable in a laboratory" and "noticeable in the bathroom mirror" is typically 4 to 6 weeks. People who quit after two weeks because "nothing is happening" are usually quitting just as the chemistry is starting to work.
Built for the post-meal window, where remineralization actually happens.
Minvelle pairs nano-hydroxyapatite with xylitol in a chewing gum designed to be used when saliva flow and pH recovery matter most: right after you eat, drink, or sip something acidic.
See the formula →The 4 stages of enamel wear and which are reversible
Most consumer content collapses enamel damage into one bucket ("worn enamel") which makes the reversibility question impossible to answer. In clinical practice, dentists think in stages, and each stage has a different prognosis. The stages below are a simplified working framework based on standard caries and erosion progression descriptions used in restorative dentistry, including the ICDAS criteria for caries assessment.
Reversible. The outermost 30 to 50 micrometres of enamel still looks intact, but the layer underneath has lost mineral content. The classic finding is that the surface looks chalky-white when air-dried because the porous subsurface scatters light differently. There is no surface defect. Under standard remineralization protocols, calcium and phosphate ions can diffuse through the surface and rebuild the lattice underneath. This is the most reversible scenario and the one where remineralization research is most consistent.
Reversible with caveats. The surface layer has begun to lose minerals as well, often from direct acid erosion (citrus, soda, reflux). The enamel becomes microscopically soft and vulnerable to mechanical wear, especially from brushing. Crucially, the rod structure is still in place: the surface has thinned, not collapsed. Remineralization can re-harden a softened surface in laboratory studies within hours of fluoride or hydroxyapatite exposure, with continued gains over weeks. The catch: brushing too soon after acid exposure (the common "wait 30 to 60 minutes" advice) can mechanically scrape away the softened surface before it has a chance to rehard, slowing the process.
Borderline. The surface has broken down at a microscopic scale, forming pits and irregularities visible under magnification or sometimes the naked eye. A dental explorer might just catch in the surface, or it might still slide over it. Bacteria can begin colonizing the pits. Some of these lesions arrest and stabilize under aggressive remineralization with high-frequency dosing. Others progress to frank cavities, especially in deep occlusal fissures or interproximal contact areas where biofilm cannot be mechanically removed. This is the zone where individual judgment matters, and where a dentist who has seen the same tooth twice in a six-month interval is better than any product.
Not reversible. A defined hole has formed. The enamel has lost volume. Bacteria are now living inside the cavity, often progressing through the dentin underneath. Remineralization products can do nothing to refill the missing volume. The clinical options are restorative: composite filling, glass ionomer, inlay, or in advanced cases an indirect restoration. Some early Stage 4 lesions can technically be "arrested" with silver diamine fluoride or sealants without active drilling, but this is a holding pattern, not a rebuild. The volume that is gone is gone.
One reason this stage framework matters: the same tooth can have different stages in different areas. The smooth surface near the gumline might be Stage 1, the contact point between two molars might be Stage 3, and a fissure on the biting surface might be Stage 4. A whole-mouth verdict ("my enamel is worn") does not predict what will respond to remineralization. Specific surfaces do.
Why visible change comes so late
Most people expect to see something within a week or two. Remineralization rarely works on that timescale, because mineral has to move back into the enamel before anything can look different.
Mineral change in early lesions is slow
Remineralization does not whiten teeth. What can change is an early white spot: when the mineral density under its surface rises, the porous, chalky patch scatters less light and can look less chalky. That requires actual crystal growth and reorganization. The diffusion of ions through the surface layer, their incorporation into existing crystallites, and the gradual reduction of porosity all take weeks of consistent exposure.
The change is also genuinely subtle, and it never changes the shade of your teeth. Bleaching changes the chromophores inside the enamel and dentin (the molecules that absorb visible light) which is a different mechanism entirely.
Remineralization is not a fix for teeth that look dull or yellow, because it never changes their shade. If you are watching early white spots, judge them over months, not weeks. If your teeth hurt, see a dentist rather than waiting on a routine.
The slow timeline also explains a common pattern in reviews of remineralization products: users see no difference after a few weeks. That is what should happen at that point. The visible change in early white spots comes later, if it comes at all, and it is always modest.
Factors that speed up or slow down the process
Two people can run the same remineralization protocol with the same product and get visibly different results six weeks in. The variability is large and frustrating. Most of it traces back to a handful of factors that either accelerate or sabotage the mineral exchange happening at the enamel surface. Some of these are within your control, others are not.
The single biggest accelerator or sabotage is what you eat and drink. Coffee sits around pH 4.8, wine around 3.5, citrus juice and many sodas around 2.5 or lower, well below the 5.5 critical threshold for enamel demineralization. Every acidic exposure resets the clock: the mouth has to neutralize back to a safe pH (typically 20 to 40 minutes for healthy saliva) before remineralization resumes. Sipping a single coffee over two hours produces more cumulative demineralization than drinking it quickly with a meal. Frequency of acid exposure matters more than total volume. Whether a straw reduces that exposure is covered in drinking through a straw: what it protects, what it can't.
Brushing aggressively, with a hard-bristled brush, or immediately after acidic intake mechanically removes softened surface enamel before it can re-harden. Dental erosion research consistently recommends waiting at least 30 minutes after acidic foods or drinks before brushing, and using a soft-bristled brush with gentle pressure. Electric brushes with pressure sensors are useful for people who tend to scrub too hard. The brushing that feels most "thorough" is often the brushing that is doing the most damage.
Saliva is the body's primary remineralization fluid: it carries calcium and phosphate, buffers acid, and clears bacteria. Unstimulated saliva flow varies considerably between individuals, and dry mouth (xerostomia) substantially slows remineralization. Common causes include antihistamines, antidepressants, blood pressure medications, sleep apnea with mouth breathing, dehydration, and aging. Sugar-free chewing (gum, xylitol mints) is a well-validated way to increase stimulated saliva flow during the critical post-meal window. A blocked nose during a cold is a common short-term cause, covered in our piece on colds, flu, cough medicine and your teeth.
Most remineralization research finds that frequency of exposure to the active ingredient matters more than concentration, a dosing principle well documented for fluoride and consistent with early nano-hydroxyapatite data too. A small dose three or four times a day generally outperforms a single high-concentration dose once a day. This is part of why chewing gum has practical advantages over toothpaste for some users: it can be used after meals when toothbrushing is impractical, extending the daily exposure window.
Younger teeth tend to remineralize faster: higher pulp blood flow, more reactive dentin, generally higher saliva volume. With age, saliva flow declines on average, dentin becomes more sclerotic (less permeable to ion exchange), and medication-related dry mouth becomes more common. Acid reflux, bulimia, and frequent vomiting (from chemotherapy, pregnancy, or other causes) create chronic acid exposure that can outpace any consumer remineralization protocol. None of this stops remineralization, it just shifts the realistic timeline.
Both fluoride and nano-hydroxyapatite have a body of evidence supporting their role in remineralization. The 2022 Clinical Oral Investigations review by Wierichs and colleagues found nano-hydroxyapatite showed comparable potential to fluoride under remineralizing conditions specifically, though the authors rated the evidence not yet conclusive and long-term head-to-head clinical caries trials are still limited. Most adults benefit from a fluoride toothpaste at minimum, with hydroxyapatite as an additional option for people who prefer a fluoride-free approach or want a non-toothpaste delivery format like gum. To choose the paste itself, our best remineralizing toothpaste guide compares seven active families, fluoride included.
When to stop self-treating and see a dentist
The hardest part of writing honestly about remineralization is that it works for some problems and does not work for others, and the failure mode of self-treating the wrong problem is a tooth that quietly gets worse. The single most important rule: any tooth pain or sensitivity that fits the patterns below is not a remineralization problem. It is a clinical problem, and the appropriate response is a dental exam.
Red flags that mean see a dentist now
This pattern often indicates a hairline fracture (a "cracked tooth"), a high bite on a filling, or an inflamed pulp. Remineralization products do nothing for cracks or pulpitis. Untreated cracks can propagate and lose the tooth.
Brief sensitivity to cold that resolves within seconds suggests dentin exposure. Pain that throbs for minutes or hours afterwards, or wakes you up at night, suggests pulpitis, which is a clinical problem that typically requires endodontic treatment.
Once you can see a hole in the tooth or a defined dark spot in a fissure, the enamel has cavitated. No product will refill it. The longer it goes, the more likely the cavity progresses into the dentin and pulp, at which point a filling becomes a root canal.
Focal, persistent sensitivity in one specific tooth often points to a specific defect (cracked tooth, leaking filling, exposed root surface, occult cavity) that needs imaging or examination.
If you have been using a remineralization regimen four times daily, controlling your acid intake, and you have seen no change after 12 weeks, the underlying problem is likely not what you assumed. An exam can clarify whether you are dealing with exposed dentin, cracks, untreated cavities, or another issue.
Periodontal problems are not enamel problems. Remineralization products do not treat gum disease, and gum recession exposes root surfaces (cementum, then dentin) that respond differently than enamel. Decay that starts on those exposed roots behaves differently too, as our guide to root cavities explains.
These are emergencies. Skip the gum aisle and call a dentist.
A Stage 3 lesion that responds to remineralization in three months is much cheaper than the Stage 4 lesion it becomes if you ignore it for nine months. Dental care is one of the few areas of medicine where early professional intervention is dramatically cheaper than late intervention. A six-month checkup is not optional infrastructure.
The general principle: remineralization products are a maintenance and early-stage tool. They are not diagnostic tools, they are not pain management, and they are not a substitute for clinical examination. The clearest framing is to think of them as part of routine oral care alongside brushing, flossing, and dental visits, not as something you escalate to when something is wrong.
Want a baseline before you start counting the weeks? Take the free 2-minute enamel quiz: 7 questions, your enamel score, and a plan that matches it. Take the quiz →
How to track your own progress
Remineralization is a slow enough process that without some form of structured tracking, it is genuinely hard to tell whether anything is changing. Memory is notoriously unreliable: most people remember the worst recent day and forget the baseline. Visual change is small enough that it can be hidden by lighting, camera angle, or time of day. A simple tracking protocol turns vague impressions into something you can actually evaluate.
Symptom diary
If you have cold sensitivity, keep a short diary while you change your routine: rate it 0 to 10 once daily after the same trigger (a sip of room temperature water, then a sip of cold water from the fridge), and note any spontaneous pain events. That is much more reliable than asking yourself once a month, and it gives your dentist something concrete. Sensitivity that persists or gets worse is a reason for an exam, not for more product.
Before and after photos in consistent lighting
For tracking cosmetic change, the trick is reducing variability. Use the same camera, the same time of day, the same room and lighting position, and the same facial expression. Natural daylight from a window is more consistent than mixed indoor lighting. Take photos with teeth dry (wipe with a tissue first) because dry enamel reveals white spot lesions and chalky patches more clearly than wet enamel. Weekly intervals are usually enough, monthly is fine for slower changes. Compare photos directly side by side, not from memory.
Dentist baseline measurements
If you are starting a serious remineralization protocol because of a known concern (white spot lesions, early erosion, post-orthodontic damage), an extra layer of tracking comes from your dentist. Many practices now use laser fluorescence devices (such as DIAGNOdent) that give a numeric measurement of mineral density at specific points, repeatable visit to visit. Some use intraoral cameras with magnification that capture surface texture in a way the naked eye cannot. Asking for a baseline at the start and a comparison at six months turns a subjective experience into a measurable one.
What to measure, what not to bother with
Worth measuring: visible chalkiness on specific teeth (yes or no, photographed), any new dark spots or pits, and, if you have it, a cold sensitivity score to show your dentist. Not worth measuring: overall "tooth whiteness" using a shade guide at home (lighting variation swamps the signal), tongue sensation of "smoothness" (highly variable, unreliable), or how often you brush (everyone overreports). The goal is to track the variables that actually correlate with what remineralization changes.
The honest expectation for three months of a remineralization routine is no dramatic change: early white spots may look a little less chalky, and tooth shade stays the same. If a product promises more than that without surgical or bleaching intervention, the product is overselling.
The arc of a remineralization routine looks something like this: for the first weeks you see nothing, which is normal; over the following months early white spots may look a little less chalky in photos; and by months 3 to 6 things level off into a stable baseline. You then maintain that baseline with continued use rather than expecting further dramatic gains. This is what "remineralization works" actually looks like in practice. It is not transformation, it is slow chemistry.
What the successful study protocols have in common
Reading across the clinical trials cited in this article, the protocols that produced measurable surface-hardness and mineral-density gains share four habits, and none of them are exotic. First, exposure frequency beat exposure dose: several short contacts with calcium, phosphate, or hydroxyapatite spread across the day outperformed one long one, which is why after-meal timing keeps appearing in the methods sections. Second, every successful protocol protected saliva. Participants with dry mouth, whether from medication or mouth breathing, consistently sat at the bottom of the response curves. Third, acid exposure was controlled during the study window; remineralizing while sipping acidic drinks all afternoon measured out close to zero net gain. Fourth, the studies ran their full length. The early weeks routinely showed nothing on the instruments, and the significant differences appeared after week four or later.
Translated into a daily routine, that means the supporting habits matter as much as the active ingredient: check the full evidence review on remineralizing gum for how chewing fits the frequency principle, vitamin D status for the systemic side of mineral supply, and the 2026 evidence ranking of remineralizing actives to choose the mechanism that fits your situation. If you are tracking progress visually, white spots are usually the first place improvement becomes visible.
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Frequently asked questions
How long until I see results from remineralizing gum?
Expect weeks to months, not days. Surface enamel hardening in laboratory remineralization studies is measured over a few weeks, depending on the model. Early white spots that fade do so over months, and the shade of your teeth does not change. The variability is high: dosing frequency, saliva flow, diet pH load, and starting condition all matter. Remineralizing gum is a slow background process, not a fast cosmetic fix.
Can severely damaged enamel be rebuilt?
No. Remineralization only works on the subsurface and surface stages where mineral has been lost but the enamel structure is still intact. Once the lesion cavitates (forms an actual hole through the enamel), the body cannot rebuild that missing volume. The protein scaffold that originally guided enamel formation, the ameloblast layer, is gone by the time teeth erupt. Cavitated lesions need professional restoration: composite, glass ionomer, inlay, or crown depending on size. Remineralization products at that point manage surrounding enamel, not the cavity itself.
What if nothing has changed after 6 weeks?
Several possibilities. Ongoing acid exposure (citrus, soda, kombucha, wine, reflux) can outpace remineralization. Early white spots often need months, and deep or long-standing ones may not change at all. If you also have sensitivity, its source may be a hairline crack, an inflamed pulp, an exposed root surface from gum recession, or a high bite point on a filling. After 6 weeks of consistent routine with no change, a dental exam is the next step, not a different gum.
Is age a factor in remineralization speed?
Yes. Younger teeth have higher pulp blood flow, more reactive dentin, and generally higher unstimulated saliva volume, all of which support faster mineral exchange. With age, saliva flow tends to decrease, dentin becomes more sclerotic (less permeable), and medication-induced dry mouth becomes more common. None of this stops remineralization, but it tends to slow it. Studies in older adults with stimulated saliva (through sugar-free chewing) show remineralization is still active, just at a more modest rate than the textbook teenage example.
Can I speed up remineralization?
To a degree. The two biggest accelerators are reducing acid exposure (cutting frequency of low pH foods and drinks) and increasing dosing frequency of remineralizing agents. Saliva is the body's primary remineralization fluid, so anything that increases flow rate (sugar-free chewing, adequate hydration, nose breathing instead of mouth breathing) helps. What does not work: doubling product concentration, brushing harder, or whitening more. Aggressive intervention often slows the process by mechanically removing the soft, partially remineralized surface layer before it can fully harden.
Build remineralization into your day, one chew at a time.
Minvelle is a nano-hydroxyapatite and xylitol gum designed to deliver active ingredients during the post-meal window when saliva flow and pH recovery matter most. Subtle, slow, consistent.
Try Minvelle →Or subscribe: 2 boxes every 4 weeks, €15.00 per box, skip or cancel anytime →
- Wierichs RJ et al., Clinical Oral Investigations, 2022. Systematic review and meta-analysis of nano-hydroxyapatite for caries prevention; similar remineralizing potential to fluoride under lab conditions, evidence not yet conclusive.
- Foundational reviews on the demineralization-remineralization balance and the critical pH threshold for enamel.
- Schlagenhauf U et al., Journal of Investigative and Clinical Dentistry, 2019. 6-month randomized trial, 150 caries-active orthodontic patients: fluoride-free microcrystalline hydroxyapatite toothpaste non-inferior to fluoride for enamel caries progression.
- Paszynska E et al., Scientific Reports, 2021. 1-year double-blind randomized trial, 207 children: hydroxyapatite toothpaste non-inferior to fluoride toothpaste for early childhood caries.
- Grocholewicz K et al., Scientific Reports, 2020. Randomized trial, 92 adults with initial approximal lesions: 6 months of nano-hydroxyapatite gel, radiographic follow-up at 1 and 2 years.
- Xylitol and reduction of Streptococcus mutans in clinical trials.
- European Scientific Committee on Consumer Safety (SCCS), Opinion on nano-hydroxyapatite in oral care, 2023.
- Ismail AI et al., Community Dentistry and Oral Epidemiology, 2007. The International Caries Detection and Assessment System (ICDAS), the staging framework for enamel and dentin caries.
- Erosion progression, post-acid brushing timing, and clinical recommendations on remineralization windows.
- SCCS, Opinion on Hydroxyapatite (nano), SCCS/1648/22, final version 21-22 March 2023
Max, Founder of Minvelle. Reads dental research daily, not a medical professional. Every Minvelle post is fact-checked against primary sources, no LLM-generated content goes live unedited. More on how this brand started.
Last reviewed: September 25, 2026 by Max, Founder of Minvelle.
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Looking for the gum itself? Nano hydroxyapatite gum, 5.7 mg per piece: dose printed on the box, 18 pieces, one a day, chew for 20 minutes.
